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Human cancer results from alterations of the genome of cells that are exposed to environmental agents that can activate oncogenes or inactivate tumor suppressor genes. In this investigation, we studied the influence of constitutive expression of heat shock protein (Hsp70) on skin cancer development in the mouse using a transgenic co-mouse model, which expresses human ras-venus genes on a keratin-14 gene-deleted background. To examine whether constitutive expression of Hsp70 exerts protective effects, we crossbred transgenic mice with a p53 mutant mouse. The type of skin tumors that develop in the mouse is dependent on the level of expression of ras-venus. These results indicate that Hsp70 can suppress the development of skin cancer in the mouse and show that such suppression is dependent on the level of transgene expression.”Hey, you!” “Back up!” “Is that really you?” “Yeah, it’s me.” “It’s me, Boshi.” “You’re Boshi, the St. Mary’s boy?” “Yes, Father.” “I’m sorry, Father!” “I’m sorry, Father.” “I didn’t mean to.” “I’m sorry.” “Forgive me, Father.” “I’m sorry, Father.” “I’m sorry, Father.” “I’m sorry, Father.” “I’m sorry, Father.” “Oh, for the love of God, Ren!” “Look at that!” “Can you believe it?” “That’s the last one!” “I wonder what happened to it?” “Well, have a look for yourself!” “Oh!” “How can you do that?” “!” “A bit of a problem, Mr. Sulu.” “Which one should I take?” “There’s a 100,000-yen reward for your capture.” “Bring me the body of the School Masters.” “I see.” “You’ll get your reward, Mr. Sulu.” “I promise.” “But don’t think for a moment that we won’t try and kill you.” “I know that, Mr. Sulu.” “But perhaps, you’d better leave it to me.” “The eyes of the dragon!” “You must be joking
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